what role may excess abdominal fat have in the development of high blood pressure?

High blood pressure is linked to excess abdominal fat, particularly visceral fat. Abdominal obesity and hypertension are linked by many physiological pathways.

Insulin resistance, which reduces cell response to insulin, is typically linked to abdominal obesity.

Chronic inflammation causes endothelial dysfunction, oxidative stress, and vascular damage, which raises blood pressure.

Adipose tissue releases adipokines. Obesity and hypertension may be linked to leptin and adiponectin imbalances.

Sympathetic activity narrows blood vessels and strengthens cardiac contractions, raising blood pressure.

Kidney sodium reabsorption increases with insulin resistance, which is typical in abdominal obesity.

Weight loss and insulin sensitivity can be achieved with regular aerobic and strength training.

Hypertension and abdominal obesity patients should contact with doctors for tailored recommendations, including lifestyle changes and medicinal therapies.

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